Community; however, may require brief inpatient care on a medical or subacute unit for stabilization/treatment of status epilepticus.
Cerebrovascular accident (CVA)/stroke
Craniocerebral trauma (acute rehabilitative phase)
Psychosocial aspects of care
Substance dependence/abuse rehabilitation
Patient Assessment Database
Fatigue, general weakness
Limitation of activities/occupation imposed by self/significant other (SO)/healthcare provider or others
Altered muscle tone/strength
Involuntary movement/contractions of muscles or muscle groups (generalized tonic-clonic seizures)
Ictal: Hypertension, increased pulse, cyanosis
Postictal: Vital signs normal or depressed with decreased pulse and respiration
Internal/external stressors related to condition and/or treatment
Irritability; sense of helplessness/hopelessness
Changes in relationships
Wide range of emotional responses
Ictal: Increased bladder pressure and sphincter tone
Postictal : Muscles relaxed, resulting in incontinence (urinary/fecal)
Food sensitivity nausea/vomiting correlating with seizure activity
Dental/soft-tissue damage (injury during seizure)
Gingival hyperplasia (side effect of long-term phenytoin [Dilantin] use)
History of headaches, recurring seizure activity, fainting, dizziness
History of head trauma, anoxia, cerebral infections
Prodromal phase: Vague changes in emotional reactivity or affective response preceding aura in some cases and lasting minutes to hours
Presence of aura (stimulation of visual, auditory, hallucinogenic areas)
Postictal: Weakness, muscle pain, areas of paresthesia/paralysis
Seizure characteristics: (ictal, postictal)
Tonic-clonic (grand mal): Rigidity and jerking posturing, vocalization, loss of consciousness, dilated pupils, stertorous respiration, excessive salivation (froth), fecal/urinary incontinence, and biting of the tongue may occur and last 2–5 min.
Postictal phase: Patient sleeps 30 min to several hours, then may be weak, confused, and amnesic concerning the episode, with nausea and stiff, sore muscles
Myoclonic: Short abrupt muscle contractions of arms, legs, torso; may not be symmetrical; lasts seconds
Clonic: Muscle contraction with relaxation resembling myoclonic movements but with slower repetitions; may last several minutes
Tonic: Abrupt increase in muscle tone of torso/face, flexion of arms, extension of legs; lasts seconds
Atonic: Abrupt loss of muscle tone; lasts seconds; patient may fall
Absence (petit mal): Periods of altered awareness or consciousness (staring, fluttering of eyes) lasting 5–30 sec, which may occur as many as 100 times a day; minor motor seizures may be akinetic (loss of movement), myoclonic (repetitive motor contractions), or atonic (loss of muscle tone). Postictal phase: Amnesia for seizure events, no confusion, able to resume activity
Status epilepticus: Defined as 30 or more minutes of continuous generalized seizure activity or two or more sequential seizures without full recovery of consciousness in between, possibly related to abrupt withdrawal of anticonvulsants and other metabolic phenomena. If absence seizures are the pattern, problem may go undetected for a period of time because patient does not lose consciousness
Complex (psychomotor/temporal lobe): Patient generally remains conscious, with reactions such as dream state, staring, wandering, irritability, hallucinations, hostility, or fear. May display involuntary motor symptoms (lip smacking) and behaviors that appear purposeful but are inappropriate (automatism) and include impaired judgment and, on occasion, antisocial acts; lasts 1–3 min. Postictal phase: Absence of memory for these events, mild to moderate confusion
Simple (focal-motor/Jacksonian): Often preceded by aura (may report deja vu or fearfulfeeling); no loss of consciousness (unilateral) or loss of consciousness (bilateral); convulsive movements and temporary disturbance in part controlled by the brain region involved (e.g., frontal lobe [motor dysfunction], parietal [numbness, tingling], occipital [bright, flashing lights], posterotemporal [difficulty speaking]). Convulsions may march along limb or side of body in orderly progression. If restrained during seizure, patient may exhibit combative and uncooperative behavior; lasts seconds to minutes
Headache, muscle/back soreness postictally
Paroxysmal abdominal pain during ictal phase (may occur during some partial/focal seizures without loss of consciousness)
Alteration in muscle tone
Ictal: Clenched teeth, cyanosis, decreased or rapid respirations; increased mucous secretions
History of accidental falls/injuries, fractures
Presence of allergies
Decreased general strength/muscle tone
Problems with interpersonal relationships within family/socially
Limitation/avoidance of social contacts
Familial history of epilepsy
Drug (including alcohol) use/misuse
Increased frequency of episodes/failure to improve
DRG projected mean length of inpatient stay: 4.4 days
May require changes in medications, assistance with some homemaker/maintenance tasks relative to issues of safety, and transportation
Refer to section at end of plan for postdischarge considerations.
Electrolytes: Imbalances may affect/predispose to seizure activity.
Glucose: Hypoglycemia may precipitate seizure activity.
Blood urea nitrogen (BUN): Elevation may potentiate seizure activity or may indicate nephrotoxicity related to medication regimen.
Complete blood count (CBC): Aplastic anemia may result from drug therapy.
Serum drug levels: To verify therapeutic range of antiepileptic drugs (AEDs).
Toxicology screen: Determines potentiating factors such as alcohol or other drug use.
Skull x-rays: Identifies presence of space-occupying lesions, fractures.
Electroencephalogram (EEG) may be done serially: Locates area of cerebral dysfunction; measures brain activity.
Brain waves take on characteristic spikes in each type of seizure activity; however, up to 40% of seizure patients have normal EEGs because the paroxysmal abnormalities occur intermittently.
Video-EEG monitoring, 24 hours (video picture obtained at same time as EEG): May identify exact focus of seizure activity (advantage of repeated viewing of event with EEG recording).
Computed tomography (CT) scan: Identifies localized cerebral lesions, infarcts, hematomas, cerebral edema, trauma, abscesses, tumor; can be done with or without contrast medium.
Magnetic resonance imaging (MRI): Localizes focal lesions.
Positron emission tomography (PET): Demonstrates metabolic alterations, e.g., decreased metabolism of glucose at site of lesion.
Single photon emission computed tomography (SPECT): May show local areas of brain dysfunction when CT and MRI are normal.
Magnetoencephalogram: Maps the electrical impulses/potential of brain for abnormal discharge patterns.
Lumbar puncture: Detects abnormal cerebrospinal fluid (CSF) pressure, signs of infections or bleeding (i.e., subarachnoid, subdural hemorrhage) as a cause of seizure activity (rarely done).
Wada’s test: Determines hemispheric dominance (done as a presurgical evaluation before temporal lobectomy).
Prevent/control seizure activity.
Protect patient from injury.
Maintain airway/respiratory function.
Promote positive self-esteem.
Provide information about disease process, prognosis, and treatment needs.
Seizures activity controlled.
Capable/competent self-image displayed.
Disease process/prognosis, therapeutic regimen, and limitations understood.
Plan in place to meet needs after discharge.
Source : http://nursingpedia.blogspot.com/2011/07/nursing-care-plan-seizure-disorders.html